These are the first REFERENCES I Found in 1967 FIRST PUBLISHED WORK ABOUT REPAIR OF MYELIN is by Bunge Mary B, Bunge RP and Ris H (1961) 
Ultrastructural Study of Remyelination in Experimental Lesion in Adult Cat Spinal Cord. J Biophys Biochem Cyto 10:67-94.  

FROM THE ABSTRACT p67; An occasional myelin sheath is first seen at 19 days; by 64 days, all axons are at least thinly myelinated. 
These observations suggest that myelin is reformed in the lesion in the same way it is first formed during normal development. 
The mechanism of myelin formation is basically similar to that proposed for peripheral nerve, and mammalian and amphibian optic nerve; it does not agree with present views on the mechanism of remyelination in adult mammalian central nervous tissue. 
FROM OBSERVATIONS p81; The neurological condition of the experimental animal begins to improve at a time when remyelination begins (19 days EM) and has returned to normal by the time most axons are at least thinly remyelinated (64 days EM). 

Foundations of Embryology  Patten Bradley M, McGraw-Hill Book Company, Inc, New York London Toronto (1958) 
One sentence from this textbook gave me one more vital hint - Cerebrospinal Conduction Paths, p351: Phylogenetically these (cerebrospinal) conduction paths in the peripheral part of the cord increase in conspicuousness concomitantly with the increasing extent to which the brain assumes a co-ordinating control over the basic reflexes which constitute the primary function of the cord ... 
MY TRANSLATION: During development, the "white matter" or myelin, in the peripheral part of the cord, becomes more obvious as the brain assumes a co-ordinating control over the basic reflexes which constitute the primary function of the cord. In other words,  movement speeds generation of myelin. 

Bunge et al suggest that myelin is reformed in the lesion in the same way it is first formed during normal development; 
Patten describes formation of myelin during normal fetal development.   
This, plus the evidence that the cats recovered without drugs, physio, counseling, supplements, special diets, or intervention of any kind, was all I needed to embark on recovery. All the details are in my book. 

Feigin I and Popoff N (1966) Regeneration of Myelin in Multiple Sclerosis. Neurol 16: 364-372. 
The title of the first paper I read about MS said it all. The authors describe the repair of myelin where the damage of  MS occurs in the central nervous system (brain and spinal cord), with myelin from the peripheral nervous system, which is unaffected by MS. Repair was observed within plaques of damage. 
FROM THE OBSERVATIONS: Nerve fibres myelinated by peripheral myelin were found within plaques in five cases of multiple sclerosis. These fibres were present singly, in small clusters, and, occasionally, in large groups. p364 
... most studies suggest that it is the nature of the neurons, and not that of the sheath cell, that determines whether the axon will or will not myelinate. p369 
FROM THE SUMMARY AND CONCLUSIONS: The regeneration of myelin may play a role in the clinical remissions of multiple sclerosis, but other factors appear to be more important. If ways were found to enhance the regeneration observed in this study, a clinically useful purpose might be served. 

More research on myelin: JOURNAL REFERENCES 
1 - Spontaneous repair of damaged myelin begins in 17-19 days. 
    [This means that myelin repair was underway long before injections began.] 

2 - Movement speeds repair. 
    [Unrestrained animals heal naturally.] 

3 - Clinical condition is not related to disease activity. 
    [Human observation is not sufficient to determine 
    peak of disease activity, or, onset of inactivity/remission.] 

Past research projects were designed 
to study damage to myelin as it is observed in multiple sclerosis, 
by using human tissue in vitro, or post mortem tissue samples. 
Studies using experimental animals, and normal controls,  fixed tissue samples 
for study with the electron microscope at regular intervals. 
Spontaneous repair of myelin was observed in mice (small mammals) in 1 week, and 
in cats and rabbits (large mammals) in 7-31 days. Humans are classed as large mammals. 
Many authors have observed that thin repair is sufficient for resumption of normal function 
in experimental animals, and, in humans. 

One study (Bunge et al, 1961) observed that the neurological condition of the experimental animal 
begins to improve at a time when remyelination begins, and has returned to normal 
by the time most axons are at least thinly repaired. 

The "re - discovery" of myelin repair in the year 2000, links repair of myelin to chemical treatment. 
The process of repair of myelin has been proven spontaneous. 
When we know this fact, we can apply it to recovery. That’s my story. 

Hans Selye warns that experimental medicine is based upon the principle that, 
if a change occurs only in those subjects receiving a certain treatment, 
then the treatment must he regarded as the cause of the change. 
Here, the important fallacy is that what you gave is not necessarily what acted. 
Errors in experimental design or procedure, play some part in almost every fallacy, 
Selye also warns of dangerous traps in scientific reasoning, 
and how easy it is to miss the discovery of what is clearly before us. 

Paul Freedman writes that society honours the re-discoverer 
more than the unexpected discoverer, not only after the discovery 
has finally been accepted, but more or less permanently. 

In the New Millennium, it seems we are still missing the discovery that is clearly behind us. 
Myelin repairs itself! Movement can initiate repair & recovery. 
 
 

For Further References  *MEDLINE®